Could gluten’s toxicity extend to the nervous system, producing symptoms identical to
classical Parkinson’s disease? A new case study adds to a growing body of research
indicating that wheat’s neurotoxicity is greatly underestimated.
A remarkable new case report describing the dramatic recovery of a 75-year-old
Parkinson’s disease patient after following a 3-month long gluten free diet reveals
the need to explore whether there is an increased prevalence of silent or symptomatic celiac
disease or non-celiac gluten sensitivity both in those afflicted with Parkinson’s disease
and the related multi-factorial neurodegenerative condition known as Parkinsonism.
Published in the Journal of Neurology,[i] the report notes that celiac disease often manifests
with only neurological symptoms, even in advanced age. This may strike the reader as surprising,
considering gastrointestinal complaints are the most commonly noticeable symptom; and yet,
when the voluminous published literature on gluten-related adverse health effects is taken into account,
so-called ‘out of intestine’ expressions of intolerance to gluten-containing grains are far more than
gut-related ones, with no less than 200 distinct adverse health effects implicated.
You can read our summary of the biological carnage exacted by this ‘king of grains’ here:
Wheat: 200 Clinically Confirmed Reasons Not To Eat It. You will notice that harm to the brain figures
high on the list. From schizophrenia to mania, autism to peripheral neuropathy, the central nervous
the system is particularly sensitive to its adverse effects.
( Please finish read by clicking on this link below.)
Other factors: To consider
https://www.cancertutor.com/budwig/ 1/3 way down the page.
Improvements from jaw alignment.
Aloe Arborescens for Parkinson’s:
In conclusion, we found that the long-term treatment with the intramuscular administration of thiamine has led to a significant improvement of motor and non-motor symptoms of the patients with PD; this improvement was stable during the time and without side effects. Our report represents an important contribution to PD therapy, although further experience is necessary to exclude the placebo effect and to confirm the present observation, with clinical, cellular, and molecular data. The aim of the future studies will be to investigate the clinical, restorative, and neuroprotective effects of the long-term treatment with thiamine in PD.
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