Breast Cancer

Breast cancer will affect 1 in 8 women during their lifetime — and it is the fourth-leading cause of cancer death in the United States.

But 8 in 8 women are being exploited by those looking to monetize the disease. They tell women to “put your breast foot forward,” to “feel for lumps; save your bumps.”

The reality of breast cancer is not a catchphrase — not even for those who seek to “save second base.” Or those who champion “big or small, let’s save ’em all!” With such pithy advertising ploys, how can we not believe “the breast is yet to come”?

The 5-year relative survival rate for breast cancer ranges from 98.8 percent (Stage 1) to 26.3 percent (Stage IV). [1]

The chances for breast cancer begin rising at age 40. Women 70 and older are those most likely diagnosed with breast cancer. The median age is 59 for African-American women, compared to 63 for whites. Fewer than 5 percent of women diagnosed with breast cancer in the United States are younger than 40.

In 2017, the estimate among U.S. women is there will be:

252,710 new cases of invasive breast cancer,
and 40,610 breast cancer deaths.
Forty thousand people — that’s about the population of Hickory, N.C. Or San Gabriel, Calif. Or Hagerstown, Md. Or Crystal Lake, Ill. Or Sherman, Texas.

Imagine all the people in any of those towns vanishing from the earth in one year. … That should be sobering.

Instead, the choice of many is to exploit in the name of raising money for cancer “research.” Among the one-liners we’ve all heard under the guise of fund-raising:

Thanks for the mammories. … Real men wear pink for the cure. … Boobs, sweat, and tears. … We are fighting to keep a breast of the competition. … Yes my boobs are fake, my real ones tried to kill me.

According to its website, up to 75 percent of the net income from a Susan G. Komen Race for the Cure stays in the community. The money funds breast cancer health education, screening, and treatment programs.

The remaining 25 percent supports Komen’s national research and training grants program.

Put another way: Sick care is more profitable than health care.
To finish this great article by the cancer tutor please click this link:
KNOW your stomach acid!!


I need 2 min. of your time!
And then again at 20:40-21:45
WHY no one should EVER have a needle biopsy done!!

Over 30 protocols!!


The above video is a MUST watch!
Minerals Magnesium & Iodine & breast cancer:

Boron for Breast Cancer
Inhabition of PI3K /Akt and mTOR with Herbs & supplements:
Herbal help for Breast Cancer BRCA 1 & 2

In summary, our data suggest that the treatment and highly metastatic BC cell lines with the physiologically relevant levels of six phytochemicals in combination causes a significant reduction in cell proliferation, motility, invasion with concomitant induction of apoptosis. Six combination treatment caused a marked suppression in proliferation, motility, and invasion of even the resistant MDA-MB-231 cells. Moreover, the study indicated that the phytochemical combination markedly inhibited the expression of the cell adhesion molecule CD44, which is a metastasis-initiating factor. CD44 is also known as a marker for BC stem cells, the only subpopulation of cancer cells that have the ability to promote new tumor formation at secondary sites and are known to have a high resistance for cancer chemo-and radiotherapies. In the present study, we did not investigate the effects of this phytochemical cocktail on BRCA1 and BRCA2. However previous studies  have shown that BRCA1 and BRCA2 are molecular targets for four of the six compounds (Indole-3-carbinol, Resveratrol, Genistein, and Curcumin) used in this phytochemical cocktail. Thus, it will be of great interest to evaluate any synergistic or additive effects of this cocktail on the expression BRCA2. Further, we previously demonstrated that I3C and RE synergize to effectively kill ovarian cancer cells , thus making this super cocktail effective against this cancer also. Future experiments include animal studies using a use xenograft model to evaluate the in-vivo toxicity and efficacy of the phytochemical super cocktail treatment to prevent and/or regress BC tumors as well as possible use of the highly up-regulated novel genes as markers to follow-up progress of therapy.
HERBAL CHEMO: ********************************************************…/
CoQ-10 better form is Ubiquinol:
HER2 Breast Cancer:

Glutamine:  THIS IS HUGE!


CoQ 10 or Ubiquinol:


Parsley has the highest amount of APIGENIN in it!

COPPER AND TNBC  ****************************             

Background: The tumor microenvironment (TME) plays a critical role in the spread of tumors. Bone marrow derived VEGFR2+endothelial progenitor cells (EPCs) and copper-dependent lysyl oxidase (LOX) are key in tumor progression. We hypothesized TM-associated copper depletion inhibits tumor metastases by reducing the number of EPCs and other copper dependent (CD) processes in the pre-metastatic niche. These results are an update with longer follow-up. Methods: Phase II study of BC pts at high risk for recurrence, defined as node+ triple negative (TNBC), stage 3 and 4 with no evidence of disease (NED) were enrolled on a trial of CD with TM. Ceruloplasmin (Cp) levels were maintained between 8-16 mg/dl for two years with an extension phase or until relapse. The primary endpoint was change in EPCs measured by flow cytometry before and during treatment. Secondary endpoints included tolerability, safety, PFS and LOXL-2 levels. Results: 75 pts received 2650 cycles of TM on primary and extension study. The median age is 51 years (range 29-66). Forty-five pts have stage 2/3 BC and 30 with stage 4 NED. TNBC pts were 48% and 40% of pts are stage 4 NED. Median Cp level decreased from 28 to 16 (p < 0.0001) after one cycle. Copper depletion was most efficient in TNBC where Cp levels dropped from 23.5 to 13 after one cycle. TM was well tolerated with grade 3/4 toxicities including: reversible neutropenia (2.3%), febrile neutropenia (0.04%), fatigue (0.2%). Five-year analysis showed a decrease in EPC’s (p = 0.004) and LOXL-2 (p < 0.001). At a median follow-up of 6.9 years, the EFS for 75 pts is 75.6%. PFS for 36 pts with TNBC is 79.2%. EFS for stage 2/3 TNBC is 90% and for stage IV TNBC is 66.7%. Conclusions: TM is safe, well tolerated and appears to affect multiple components of the TME creating an inhospitable environment for tumor progression especially in high risk patients such as TNBC. Randomized trials are warranted, especially in patients at high risk for relapse. Clinical trial information: UL1TR000457.

Natural ways to lower copper quickly are TM

& if a dr. won’t write a script for it start her on





© 2017 by American Society of Clinical Oncology
THIS IS HUGE:…/2019/05/190513112242.htm
Is this a solution to TNBC??
Her2 & Triple negative
Video below 1 min. mark for TNBC

Milk thistle for TNBC:
___________________ (organic dried ginger)
Bee Venom Aggressive against Breast Cancer (even TNBC) ***********************
In vitro and in vivo assays have demonstrated that propolis activated monocytes/macrophages and neutrophilsincreasing their microbicidal activity. It enhanced the lytic activity of natural killer cells against tumour cells.
Liposomal curcumin & resveratrol for TNBC
Ivermectin for TNBC !!!!

Recent data support the capacity of ivermectin to kill mice and human triple-negative breast cancer (TNBC) cells through an inflammatory and immunogenic mechanism and demonstrate that tumor cells release ATP and suffer from ICD mediated by increased P2X4/P2X7-gated pannexin-1 channel opening, which causes cell death.Jan 21, 2020


 Ivermectin, a potential anticancer drug derived from an …

Triple-negative breast cancer (TNBC) refers to cancer that is negative for estrogen receptor, progesterone receptor, and human epidermal growth factor receptor 2(HER2) and is the most aggressive subtype of breast cancer with the worst prognosis. In addition, there is also no clinically applicable therapeutic drug currently [34,35].



mately 50 years[7]. In recent years, niclosamide was found a potential anticancer drug against various cancers including human lung cancer[8,9], ovarian cancer[10], prostate cancer, head and neck [11,12] cancer[13], osteosarcoma[14] and breast cancer[15,16] both in vivo and in vitro. Niclosamide is a multi-targeted anticancer drug that may target


Omega -3’s for breast cancer Her2 + & TNBC


More on Iodine for breast health:


Image may contain: text
 German New Medicine:

For more on this topic visit:

TNBC is mentioned in the studies at 1:07 mark

Recent updates:
FAS inhibitors
Fig. 2

NORI Protocol – Nutritional Oncology Research Institute


NORI has researched and developed a unique approach to cancer therapy that is very simple, … An important feature of the NORI protocol is the synergy created between the methionine … More videos on YouTube. Share … Methionine Deprivation Suppresses Triple-Negative Breast Cancer Metastasis In Vitro and In Vivo.

Glutamine:  THIS IS HUGE!
Inhibition of P13K/AKt/mTOR Signaling by NATURAL products:

Problems with chemo for cancer:
Thyroid meds & breast cancer:

What if everything doctors told you about breast cancer was wrong?

Toxicity of thieves oils to mcf-7 & mda-mb-231 breast cancer cells: *************
Natural treatments for breast cancer:


Breast Cancer & Walnuts:
Why NOT Tamoxifen? Because fewer side effects  with natural.

Tamoxifen does, however, increase the risk for uterine cancer (endometrial cancer and uterine sarcoma). Still, the overall risk of uterine cancer in most women taking tamoxifen is low, and studies have shown that the benefits of this drug in treating breast cancer are greater than the risk of second cancer.

Natural Alternatives!!
DIM / IC3 / Flax seeds & the oil / organic soy red clover / yam

Second Cancers After Breast Cancer | Journey Forward › second-cancers-caused-breast-cancer-treat


Tamoxifen VS Seaweed:    SEAWEED for the WIN!! PLUS LIVER protection


Here is the bottom line:

Results from this and other laboratories support the hypothesis that white button mushrooms may be an important dietary constituent for reducing the incidence of hormone-dependent breast cancer in women. Prevention strategies involving mushrooms are readily available, affordable, and acceptable to the general public. The dosage used in the in vivo trial is also physiologically relevant. A typical conversion factor for converting mouse to human dosage of chemotherapeutic agents is 25, calculated based on body surface area (53). Thus, according to our preliminary results generated from MCF-7aro-derived tumor formation in nude mice, consumption of 100 g of mushrooms per day would be sufficient to suppress breast tumor growth in women. As this is a pharmacologic dosage that inhibits the growth of established tumor cells in vivo, it is feasible that a lower intake of mushroom ingested regularly as a routine dietary constituent would be effective in preventing the initiation of breast tumors in an average woman. The information gained from our study can aid in the design of more highly developed and effective breast cancer prevention strategies involving dietary constituents such as mushrooms. SOURCE:

More on seaweed for cancer:
“Fucoidan was reported to enhance the activity of natural killer (NK) cells, which have anticancer activity []. The effects of crude fucoidan extracted from Fucus vesiculosus on the growth of breast cancer have been determined in vitro and in vivo []. Crude fucoidan significantly reduced the number of viable 4T1 cells (a mouse tumor cell line used as a model of highly metastatic breast cancer), enhanced apoptosis, and down-regulated the expression of vascular endothelial growth factor (VEGF). The mechanisms thought to be responsible for these fucoidan-mediated effects are inhibition of the expression of Bcl-2 (Bcl-2 preserves the mitochondria integrity), survivin, extracellular signal-regulated kinases (ERKs), and VEGF, and an increase in caspase-3 activation.”         AND Conclusion:

Many studies have explored the use of seaweed in the fight against several diseases, including colorectal and breast cancers. Various therapeutic compounds from seaweed are able to induce apoptosis through different pathways and molecular mechanisms. Several studies indicated that fucoidan was able to induce apoptosis, inhibit angiogenesis, and suppress lung metastasis of breast cancer in vitro and in vivo [,,,,,]. Furthermore, fucoidan inhibited growth and induced apoptosis of HT-29 colon cancer cells []. Laminarin induced apoptosis through the Fas and IGF-IR signaling pathways and through the intrinsic apoptotic and ErbB pathways []. This review highlights the importance of seaweed in the fight against colorectal and breast cancer. SOURCE:

Buy Fucoidan:
German New Medicine on metastasis:

Chart of normal and optimal thyroid levels values: Thyroid ...


Converting Units (IU) Of Vitamin D To Micrograms (mcg)

May 5, 2022Vitamin D 1,000 IU = 25 mcg (0.025 milligrams) Vitamin D 2,000 IU = 50 mcg (0.050 milligrams) Vitamin D 5,000 IU = 125 mcg (0.125 milligrams) Vitamin D 10,000 IU = 250 mcg (0.250 milligrams) Vitamin D 50,000 IU = 1,250 mcg (1.25 milligrams)

Suggested daily supplements for most cancers by some people:
has not been approved by the FDA. (not meant to treat disease)
******* This for people NOT wanting to do the Budwig Protocol
Autophagy natural inhibitor:
diverse phytochemicals derived from natural sources, such as curcumin, ursolic acid, resveratrol, thymoquinone, and γ-tocotrienol, also have attracted attention as promising autophagy modulators with minimal side effects.  SOURCE:

Ashwagandha for BREAST CANCER:


****************** FENBENDAZOLE TESTIMONIALS  ******************************

THIS IS A NON-BUDWIG protocol!! Used by some people.

 Iron: ZERO, supplementing iron is dangerous and should only be done by a specialist.

Melatonin :The suggested cancer fighting dose for stage 4 cancer patients is 60mg 3 to 6 times a day and 180mg each night.

Beta Carotene 30,000 IU -50.000 IU

B Complex 50 to 100 mg

B-12 liposomal 1,000 mcg = 1mg (early in the day as to NOT affect sleep)

Vitamin C 18 to 40 grams a day

Vitamin E 300 IU in the form of E succinate

Selenium 600m 3-5 Brazil nuts would be an excellent option

Zinc 60 mg

Vitamin D3 at 4,000 to 6,000 IU (encourage you to take it with K2)

Vitamin K2 at 100mcg to 300mcg a day

Coenzyme Q10 at 300 mg

A combination of Curcumin 3,000 mg with Bioperine 15

Additional known cancer fighting supplements:

4 parts IP6 and part inositol, provides the body with building material to also make IP2, 3,4,and 5

Beneficial dosing 500mg, cancer fighting dosing 4 to 6 Grams a day

R-ALA : Beneficial dosing 100mg, cancer fighting dosing 1200 to 1800 mg a day min mark 13:30-14:35

NAC : Beneficial dosing 500mg, cancer fighting dosing 3 grams a day

MSM : Beneficial dosing 500mg, cancer fighting dosing 3 grams to 30 grams a day. Obviously mega-dosing is not for everyone.

Magnesium: Beneficial dosing 500mg, cancer fighting dosing 1 to 3 grams a day

Organic Coconut Oil: 3 to 6 tablespoons a day, usually melted in food and warm drinks.

Quercetin with Bromelain 600 mg 2 – 3  X a day

Berberine 500 mg 2-3 X a day

Artemisinin 400 mg 2 X a day (mixed with manuka honey 1/2 tsp. for 800-900 mgs.)

Cold-pressed flax seed oil 2 Tablespoons daily.

Black seed oil 2 tsp a day

Triphala 1 teaspoon a day

LDN 1.5 mg to 4.5mg a day
For more info on these supplements look here:
Budwig Protocol:


DIM / IC3 / ZINC / Stinging nettle root tea / Passion flower / Calcium L- Glucarate / instead of Tamoxifen******
Iodine for cancer:*******************************************************;     **************                      
Cat’s claw
Methylene blue:
Natural Products as aromatase inhibitors:
PDF file for essential oils:
Flax seeds: See cancer superfoods link under flax seeds
Ruth Lupu Ph D Cancer Researcher: **
Fenbendazole & supplements a cancer cure?   ***************************
For much more on this subject look here:

Repurposed drugs – Disulfiram (Antabuse)
Stress & negative thoughts:

German New Medicine:

Please listen from min. mark 12 to 16

min mark 39:30 – 41:55 for dr. Sherry Tenpenny below:

MSM for breast cancer:

Fungating tumors:

Fungating tumors treated with manuka honey & golden drops liquid artemisinin OR black seed oil!,-yeasts-and-parasites
Castor oil packs:
ORGANIC DRIED GINGER (6-shogaol) › 26355461 › 25686711 › 6shogaol › 6shogaol.html

Ancient Egyptians were the first to mention castor oil as a medicine and since then this oil, also known as Palma Christus, has been used as a folk medicine .
Castor oil packs had been highly popularized by Edgar Cayce, “The Sleeping Prophet”, who recommended, in particular to eradicate tumors near the breast surface. The seed oil from the castor bean Ricinus communis is very rich in Δ-12-hydroxy-9-octadecenoic acid (ricinoleic acid, about 90 %,), and contains as minor components phenolic compounds, such as p-coumaric acid, ferulic acid, o-coumaric acids, syringic, cinnamic, chlorogenic, neochlorogenic, and gallic acids .
Quite a few of triglycerides of R. communis containing ricinoleic acid, have as major components triricinolein and, in addition, diricinoleo-triglycerides with ricinoleic acid at the 1- and 3-positions .
Castor oil is hydrolyzed in the small intestine by pancreatic enzymes, which results in the release of glycerol and ricinoleic acid, although 3,6-epoxyoctanedioic acid, 3,6-epoxydecanedioic acid, and 3,6-epoxydodecanedioic acid also appear metabolites. Castor oil and ricinoleic acid easily penetrate deep into the skin and they enhance the trans-dermal penetration of other chemicals.
Bioactive di- and triacylglycerides of ricinoleic acid
According to estimates of the World Health Organization, 80 % of the world population, especially those living in Asia, Latin America and Africa, still relies on herbal medicine
Castor oil packs are a cheap effective treatment to try for breast cancer .
Ivermectin for killing Cancer stem cells!

Figs or potatoes for breast lumps:

Ways to Inhibit IGF-1

If you have high IGF-1 levels, you might want to focus a bit more on things that inhibit IGF-1, especially if you’re genetically predisposed to cancer.


Flavonoids in Cancer and Apoptosis – NCBI › pmc › articles › PMC6357032
by M Abotaleb – ‎2019 – ‎Cited by 13 – ‎Related articles

Dec 28, 2018 – Flavonoids have gained importance as anti-cancer agents and have shown great ….. Galangin in combination with berberine synergistically led to cell cycle …. Chrysin was reported to be the most potent flavonoid functioning in the …… Liu R.H. Nutrition, and cancer potential synergy of phytochemicals in …

Effects of berberine on proliferation, cell cycle distribution and … – NCBI › pmc › articles › PMC4439447
by E Barzegar – ‎2015 – ‎Cited by 38 – ‎Related articles

Conclusion: Berberine alone and in combination with doxorubicin inhibited cell proliferation, induced apoptosis and altered cell cycle distribution of breast cancer cells. Therefore, berberine showed to be a good candidate for further studies as a new anticancer drug in the treatment of human breast cancer.

Cervical Cancer:
Turkey Tail & or Reshi mushrooms: › science › article › pii › B9780128190012000231 The role of microbial pathogens in cancer … – ScienceDirect HPV is a group of about 150 interrelated viruses (Robertson, 2012), and virtually the cause of all cervical cancer, with HPV 16 and 18 responsible for about 70% and HPV 16 for 85% of anal cancer cases. HPV 16 and 18 cause 50% of vulvar, vaginal, and penile cancers (Watson et al., 2008, Bruni et al., 2019). HPV types 16, 18, 34, or 35 are …
Alma powder:




In conclusion, this study clearly is an advancement of an earlier report of curcumin nanoformulation tested against cancer cells. Our findings show that PLGA based Nano-CUR significantly inhibits growth of cervical cancer cells and regulates the expression of miRNAs and various oncogenic and tumor suppressor proteins associated with cervical cancer (Fig. 7). In vivo experiments show that Nano-CUR is efficacious in reducing the tumor burden. Therefore, Nano-CUR may be a novel chemo-preventive and therapeutic modality for the management of cervical cancer.  SOURCE:

Artemisinin: › pmc › articles › PMC6637696

Olive Leaf Extract:
Budwig protocol:
Cervical cancer is the second most frequent cancer in women worldwide, yet in Turkey it ranks only ninth, which is 2 to 5 times lower than in Europe and North America. Although the reasons for this difference surely involve a combination of factors, including sociocultural differences, lack of population-based screening programs, or a lower human papillomavirus (HPV) prevalence rate in Turkey, it has been suggested that the low incidence of cervical cancer in Turkey correlates with its boron-rich soil. HPVs are the main cause of cervical cancer. HPV-16 and HPV-18 cause approximately 95% of all cervical cancers, and boron interferes in the life cycle of HPV.

Download scientific diagram | Effects of 6-shogaol on cervical cancer cell proliferation, apoptosis, and cell-cycle arrest. (a) HeLa cells were treated with 0.1% DMSO, 5, 10, 20, 40, and 80 μM 6 …


Ovarian Cancer: › 25544381
This study found that ivermectin inhibited OC migration, and that ivermectin-mediated lncRNA-EIF4A3-mRNA axes were the potential mechanism in OCs. The optimized three-lncRNA signature model (ZNRF3-AS1, SOS1-IT1, and LINC00565) provided a good assessment system to significantly associate survival risk with the expression profile of these three lncRNAs. These findings are the precious resource and have practical significance for drug redirecting of ivermectin in OC treatment for personalized drug therapy and prognostic assessment towards its PPPM practice.
Schizandra Berry:

mately 50 years[7]. In recent years, niclosamide was found a potential anticancer drug against various cancers including human lung cancer[8,9], ovarian cancer[10], prostate cancer, head and neck [11,12] cancer[13], osteosarcoma[14] and breast cancer[15,16] both in vivo and in vitro. Niclosamide is a multi-targeted anticancer drug that may target


For much more on Melatonin:

Cervical Cancer:

Artemisinin: Could It Be a Cancer Treatment? – WebMD

Advanced cervical cancer: Doctors treated 10 women with a form of artemisinin for 28 days. All 10 went into remission and saw symptoms like pain and vaginal discharge go away. Risks



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