Heart Disease

By Dr. Mercola

The video above, “The Fat Emperor: Insulin Versus Cholesterol,” features Ivor Cummins, a biochemical engineer with a background in medical device engineering and leading teams in complex problem-solving.

In 2013, Cummins ran into health problems. His serum ferritin was very high (which is a potent risk factor for heart disease), as were his liver enzymes. After consulting with three different doctors, he realized none of them really understood the root cause of these problems, or how to address them.

As a result, he delved into the medical literature, found the problem and reversed his abnormal test results. He also dropped 35 pounds in the process.

Eventually, he got more involved in health and began giving lectures such as this one, which was presented at the Low Carb USA Keto Getaway1 in Florida this past January. He also has a website, thefatemperor.com,2 where he notes:

“I refer primarily to the ‘diet-heart’ hypothesis, which proposed that dietary saturated fat elevated blood cholesterol, and the latter drove heart disease mortality like nothing else. 

The evidence at the time was loose correlation, certainly not causation, and seems almost laughably naïve in retrospect.

However, the tenaciousness of this flawed hypothesis has turned out to be no laughing matter, condemning millions to the misery of obesity, type 2 diabetes and an extraordinary range of inflammatory diseases.

The factors that conspired to perpetuate the flawed hypotheses were many: academic and research community hubris, political forces, economic imperatives, profiteering from the food and pharmaceutical industries, and the groupthink psychology that underpins the worsening ‘diabesity epidemic.

After 25 years in technical/management positions with a personal specialty in complex problem solving, I have been inspired to … bring an engineering-style approach to the current situation.”
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WHY ISN’T THIS FRONT PAGE NEWS ABOUT BORN & HEART ATTACKS??
https://www.nature.com/articles/s41598-020-73864-w?
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https://www.naturalmedicinejournal.com/journal/going-beyond-hype-hydrogen-water

We previously reported that molecular hydrogen-rich water (HW) has antioxidant and anti-inflammatory effects in numerous diseases. Here, we used LDL receptor-deficient mice fed a high-fat diet …
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PLEASE READ THIS STUDY!!
https://pubmed.ncbi.nlm.nih.gov/30198808/
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https://sayerji.substack.com/p/nattokinase-reverses-heart-diseasebut

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A must watch video!!!

 

 

 

  1. Feb 17, 2024A 2017 paper also found NAC has potent thrombolytic effects, meaning it breaks down blood clots once they’ve formed. According to the authors, “NAC is an effective and safe alternative to currently available antithrombotic agents to restore vessel patency after arterial occlusion.”
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AHA/ASA Journals

https://www.ahajournals.org › doi › 10.1161 › CIRCULATIONAHA.117.027290

Background:Platelet cross-linking during arterial thrombosis involves von Willebrand Factor (VWF) multimers. Therefore, proteolysis of VWF appears promising to disaggregate platelet-rich thrombi and restore vessel patency in acute thrombotic disorders such as ischemic stroke, acute coronary

Georgia Institute of Technology

https://www.gatech.edu › news › 2024 › 06 › 18 › novel-use-existing-drug-could-significantly-cut-heart-attack-risk

Jun 18, 2024Breaking Down Blood Clots Bresette and Ku sought to break down VWF proteins using a drug already on the market, N- acetyl cysteine ( NAC ), typically used to treat acetaminophen overdose. Earlier researchers had tried using NAC to break down clots after formation, but Ku’s team wanted to stop clots before they even started.
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Watch video directly below with face book link
https://www.facebook.com/reel/292073330654219

May be an image of text that says 'Flashback Friday: Four Nuts Once Month Watch later Share Brazil Nuts A Single Consumption of High Improves Lipid Profile of Healt Serum LDL-c levels were significantly starting at hours after the ingestion of nuts within the groups receiving Elisângela Colp 20 or 50g and reached steady evel 48 hours Brenner Reetz, Marta Maria Medeiros Frescura Duarte, Edson IrineuMuller,' Aline Lima Hermes Muller, Erico Marlo Roger Vagner, and மDட 100 Department stry, 97105900 Santa Maria, RS, Department ofNutrition, 90 Background. This study healthy subjects enrolled ofdifferent serving (UFSM). 70 VIDEOS Basal 3h 6h Brazil nut volunteers. Methods. Ten'

HIGH TRIGS & Apo B A MAJOR RISK FACTOR:

https://www.healthline.com/nutrition/foods-to-lower-triglyceride-levels
https://entirelynourished.com/15-foods-to-lower-triglycerides-and-6-to-avoid/

The Cholesterol Conundrum
The vast majority — about 80 percent — of the cholesterol in your body is made by your liver. The remaining 20 percent comes from your diet. If you consume less, your body will compensate by making more, and vice versa.

Contrary to popular belief, cholesterol is a crucial molecule necessary for optimal health, and not nearly the damaging culprit it’s been made out to be.

Since cholesterol is a fatty substance, it does not travel well through your water-based bloodstream. Hence it is encapsulated in a lipoprotein. Cummins likens the very-low-density lipoprotein (VLDL) your liver makes to a boat that shuttles not only cholesterol but also triglycerides through your bloodstream to your tissues.

The VLDL will dock onto receptors in your muscle tissue, where it releases triglycerides to be used for energy. Cummins accurately notes that eating fat is not the cause of high triglycerides.

If your triglycerides are high, it means you’re eating too many net carbohydrates, because it’s actually sugar that causes triglycerides to rise, not dietary fat.

Once the VLDL has dropped off the triglycerides to be burnt for energy (or stored as fat if you’re not using the energy due to inactivity), the VLDL becomes a low-density lipoprotein (LDL), which in conventional thinking is a “bad” kind of cholesterol.

High-density lipoprotein (HDL) is colloquially known as “good” cholesterol, and the HDL is indeed beneficial in that it acts as a master manager, helping protect the LDL against oxidation and transport triglycerides and cholesterol in and out of the VLDL.

In a healthy person, the LDL will be reabsorbed by the liver after about two days, where it gets broken up and recycled. This is a beautiful system; alas, it is one that can be disrupted if you’re eating too many unhealthy foods.

As a general rule, a high-sugar diet will cause damaged LDL to rise, beneficial HDLs to drop, triglycerides and, often, total cholesterol to rise. All of these are conventional indicators of atherosclerosis or inflammation in your arteries that can precipitate a heart attack.
For the rest of Dr. Mercola’s great read use this link:
https://articles.mercola.com/sites/articles/archive/2017/02/18/insulin-resistance-causes-heart-disease.aspx
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The screenshot below this video is from min mark 8:10

https://greenmedinfo.com/blog/how-clean-your-arteries-one-simple-fruit       POM juice
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What doctors do NOT KNOW or won’t tell you if they did!!
https://pubmed.ncbi.nlm.nih.gov/16797868/
https://men-elite.com/2017/11/16/taurine-anti-anxiety-pro-androgens-increases-gh-lowers-prolactin-2/
https://www.drlamcoaching.com/blog/taurine-supplement/
https://drhealthbenefits.com/vitamin-supplement/taurine/health-benefits-of-taurine

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THEY KNEW THIS 8 YEARS AGO!!!!!!!!!!!
https://www.sciencedaily.com/releases/2014/11/141107154651.htm

  • https://www.youtube.com/watch?v=8ipbkwzyO_8Cardiovascular: The results which were the most startling was the number of open-heart procedures which had been scheduled and were subsequently canceled when the individual’s EKG returned to normal. This happened in as little as six weeks of ingesting Organic Sulfur. 54 cases like this have been reported. Our blood vessels also regenerate, and we believe that these canceled surgical procedures could be an example of such regeneration. The group has also seen the reduction of scar tissue, high blood pressure, and the breakdown of calcium plaque in the arteries. Thus, we believe organic sulfur would be likely to benefit Alzheimer’s sufferers as well.
    Source: https://www.life-enthusiast.com/articles/the-sulfur-study/

No photo description available.

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4252213/

WOW, this is huge:
https://www.ncbi.nlm.nih.gov/pubmed/28065862
https://www.salernocenter.com/2015/03/vitamin-c-lysine-natural-plaque-reducer/

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3361290/
https://books.google.com/books?id=1ufV239YnocC&pg=PA129&lpg=PA129&dq=andrew+saul+heart+disease+and+reducing+plaque+with+vitamin+C+%26?&source=bl&ots=-S-twVGMUz&sig=ACfU3U0t_YdKm4JavgjivHWWIytOiQWtXA&hl=en&sa=X&ved=2ahUKEwj52M_ogfLlAhWhY98KHRGlB20Q6AEwB3oECAYQAg#v=onepage&q=andrew%20saul%20heart%20disease%20and%20reducing%20plaque%20with%20vitamin%20C%20%26%3F&f=false
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3750440/
Heart disease mentioned at 19:45-22:15

SPECIAL SECTION ON TREATING CHF NATURALLY:
ZZZZZZZZZZZZZZZZZZZZZZZZZZZZZZZZZZZZZZZZZZZZZZZ

The discovery that taurine is an effective therapy against congestive heart failure led to the study of taurine as a therapeutic agent against other disease conditions. Today, taurine has been approved for the treatment of congestive heart failure in Japan and shows promise in the treatment of several other

https://pubmed.ncbi.nlm.nih.gov/28065862/
https://www.lifeextension.com/magazine/2021/2/astaxanthin-heart-health
https://www.uofmhealth.org/health-library/hn-1193009
https://www.lifeextension.com/magazine/2003/12/report_heart/Page-01
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5959283/
https://www.drsinatra.com/natural-treatments-for-congestive-heart-failure

33:30 min. mark below:

https://www.greenmedinfo.com/blog/clogged-arteries-five-ways-garlic-can-help1

https://pubmed.ncbi.nlm.nih.gov/28065862/
https://www.sciencedirect.com/science/article/abs/pii/S0378517312010241
https://onlinelibrary.wiley.com/doi/full/10.1002/ptr.6282

https://nootropicsexpert.com/berberine/

You can improve the absorption of Berberine by taking it with the sodium caprate that comes in coconut oil. Take your nootropic stack containing Berberine with a tablespoon of unrefined coconut oil and problem solved. It’s best to split your dose so you’re taking 500 mg of Berberine 3-times per day. Preferably about 30 minutes before meals …
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https://www.youtube.com/watch?v=o3KRb65a7aU

PLEASE WATCH 5 min. mark till 7:40!

PAC’s Treated Naturally!!

Natural Treatment Remedies for Premature Atrial Complexes


https://pubmed.ncbi.nlm.nih.gov/16797868/

Make sure you talk with your doctor before making changes.
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STROKE:

https://www.youtube.com/watch?v=dat1NCD5lSM%5Bembed%5Dh3strongSTROKE:%5Bembed%5Dhttps://youtu.be/iBDYdTaMUTg?t=6

A FIB Treated Naturally!
https://drafib.com/blog/ultimate-guide/

Atrial Fibrillation (+ 6 Natural Ways to Help Treat ‘A-Fib’ Symptoms)


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Supplements for A-FIB:
Curcumin:

I think curcumin is a better option without the side effects!
Curcumin, the golden spice in treating cardiovascular diseases
Recent studies have demonstrated its potent activities in modulating multiple signaling pathways associated with cellular growth, proliferation, survival, inflammation and oxidative stress. The cardiovascular protective properties of curcumin in CVDs have been fully illustrated in numerous studies.
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https://www.ncbi.nlm.nih.gov › pmc › articles › PMC6369319
The effects of curcumin on the prevention of atrial and ventricular …
Curcumin has anti-inflammatory and anti-oxidant effects and thereby, it may reduce cardiovascular complications. This randomized controlled trial aimed to investigate the effects of curcumin on the prevention of atrial and ventricular arrhythmias and heart failure in patients with unstable angina. Materials and Methods:
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Curcumin has received worldwide attention for its multiple health benefits, which appear to act primarily through its anti-oxidant and anti-inflammatory mechanisms. These benefits are best achieved when curcumin is combined with agents such as piperine, which increase its bioavailability significantly. Research suggests that curcumin can help in the management of oxidative and inflammatory conditions, metabolic syndrome, arthritis, anxiety, and hyperlipidemia. It may also help in the management of exercise-induced inflammation and muscle soreness, thus enhancing recovery and subsequent performance in active people. In addition, a relatively low dose can provide health benefits for people that do not have diagnosed health conditions.  SOURCE:
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5664031/
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TAURINE for A – FIB: (It is also used a a life extension supplement)
https://www.sciencedaily.com/releases/2023/06/230608195654.htm
https://www.webmd.com/heart-disease/atrial-fibrillation/afib-supplements
https://pubmed.ncbi.nlm.nih.gov/16797868/

Click to access taurine.pdf

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CAC SCORE:

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ALCOHOL’S effect on the HEART:

Long-term alcohol use decreases myocardial protein expression and synthesis and accelerates protein degradation in the myocardium (). This in turn disrupts myocardium function, including contraction and relaxation of the cardiac walls, impairing the heart’s ability to pump blood. Using mass spectrometric–based proteomic analysis in an animal model,  found that long-term alcohol consumption was associated with decreases of 30 to 54 percent in cell-scaffolding proteins (myofibrillar α-myosin and actin) and mitochondrial proteins (mitochondrial dehydrogenases and electron transport proteins), glycolytic enzymes (glycogen phosphorylase and alpha-enolase), and fatty acid metabolism proteins (fatty acid transport protein and LCFA acyl-CoA ligase). These investigators also found decreases in peroxiredoxin 5, antioxidant protein 2, and glutathione transferase 5—important antioxidant enzymes whose cardiovascular protective functions still are not fully understood. For example, some findings suggest an inverse role between peroxiredoxin 5 and stroke severity. During a severe stroke, peroxiredoxin 5 is consumed and its production impaired ().

Other ethanol-induced changes may be related to enzymes that modulate protein synthesis and/or breakdown (e.g., ubiquitine-ligases). Several reports suggest that ethanol-induced decreases in myocardial protein synthesis may be mediated in part by decreased activity of an enzyme called mammalian (or mechanistic) target of rapamycin (mTOR) (). mTOR regulates cell growth, proliferation, motility, and survival; protein synthesis; and transcription (). Decreases in mTOR activation may play a role in reduced myocardial protein synthesis, ventricular wall thinning, and dilation.  SOURCE:
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5513687/

Controlling SVT:

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