Breast cancer will affect 1 in 8 women during their lifetime — and it is the fourth-leading cause of cancer death in the United States.
But 8 in 8 women are being exploited by those looking to monetize the disease. They tell women to “put your breast foot forward,” to “feel for lumps; save your bumps.”
The reality of breast cancer is not a catchphrase — not even for those who seek to “save second base.” Or those who champion “big or small, let’s save ’em all!” With such pithy advertising ploys, how can we not believe “the breast is yet to come”?
The 5-year relative survival rate for breast cancer ranges from 98.8 percent (Stage 1) to 26.3 percent (Stage IV). 
The chances for breast cancer begin rising at age 40. Women 70 and older are those most likely diagnosed with breast cancer. The median age is 59 for African-American women, compared to 63 for whites. Fewer than 5 percent of women diagnosed with breast cancer in the United States are younger than 40.
In 2017, the estimate among U.S. women is there will be:
252,710 new cases of invasive breast cancer,
and 40,610 breast cancer deaths.
Forty thousand people — that’s about the population of Hickory, N.C. Or San Gabriel, Calif. Or Hagerstown, Md. Or Crystal Lake, Ill. Or Sherman, Texas.
Imagine all the people in any of those towns vanishing from the earth in one year. … That should be sobering.
Instead, the choice of many is to exploit in the name of raising money for cancer “research.” Among the one-liners we’ve all heard under the guise of fund-raising:
Thanks for the mammories. … Real men wear pink for the cure. … Boobs, sweat, and tears. … We are fighting to keep a breast of the competition. … Yes my boobs are fake, my real ones tried to kill me.
According to its website, up to 75 percent of the net income from a Susan G. Komen Race for the Cure stays in the community. The money funds breast cancer health education, screening, and treatment programs.
The remaining 25 percent supports Komen’s national research and training grants program.
Put another way: Sick care is more profitable than health care.
To finish this great article by the cancer tutor please click this link:
A MUST WATCH!!
Inhabition of PI3K /Akt and mTOR with Herbs & supplements:
Herbal help for Breast Cancer BRCA 1 & 2
In summary, our data suggest that the treatment and highly metastatic BC cell lines with the physiologically relevant levels of six phytochemicals in combination causes a significant reduction in cell proliferation, motility, invasion with concomitant induction of apoptosis. Six combination treatment caused a marked suppression in proliferation, motility, and invasion of even the resistant MDA-MB-231 cells. Moreover, the study indicated that the phytochemical combination markedly inhibited the expression of the cell adhesion molecule CD44, which is a metastasis-initiating factor. CD44 is also known as a marker for BC stem cells, the only subpopulation of cancer cells that have the ability to promote new tumor formation at secondary sites and are known to have a high resistance for cancer chemo-and radiotherapies. In the present study, we did not investigate the effects of this phytochemical cocktail on BRCA1 and BRCA2. However previous studies 46–48 have shown that BRCA1 and BRCA2 are molecular targets for four of the six compounds (Indole-3-carbinol, Resveratrol, Genistein, and Curcumin) used in this phytochemical cocktail. Thus, it will be of great interest to evaluate any synergistic or additive effects of this cocktail on the expression BRCA2. Further, we previously demonstrated that I3C and RE synergize to effectively kill ovarian cancer cells 27, thus making this super cocktail effective against this cancer also. Future experiments include animal studies using a use xenograft model to evaluate the in-vivo toxicity and efficacy of the phytochemical super cocktail treatment to prevent and/or regress BC tumors as well as possible use of the highly up-regulated novel genes as markers to follow-up progress of therapy.
HERBAL CHEMO: ********************************************************
COPPER AND TNBC ****************************
Her2 & Triple negative
Liposomal curcumin & resveratrol for TNBC
EGCG for TNBC:
Omega -3’s for breast cancer Her2 + & TNBC
More on Iodine for breast health:
German New Medicine:
For more on this topic visit:
TNBC is mentioned in the studies at 1:07 mark
NORI Protocol – Nutritional Oncology Research Institute
Glutamine: THIS IS HUGE!
Inhibition of P13K/AKt/mTOR Signaling by NATURAL products:
Problems with chemo for cancer:
Thyroid meds & breast cancer:
VIDEO ON DCIS:
What if everything doctors told you about breast cancer was wrong?
Toxicity of thieves oils to mcf-7 & mda-mb-231 breast cancer cells: *************
Natural treatments for breast cancer:
Breast Cancer & Walnuts:
Why NOT Tamoxifen? Because fewer side effects with natural.
Tamoxifen VS Seaweed: SEAWEED for the WIN!! PLUS LIVER protection
More on seaweed for cancer:
“Fucoidan was reported to enhance the activity of natural killer (NK) cells, which have anticancer activity . The effects of crude fucoidan extracted from Fucus vesiculosus on the growth of breast cancer have been determined in vitro and in vivo . Crude fucoidan significantly reduced the number of viable 4T1 cells (a mouse tumor cell line used as a model of highly metastatic breast cancer), enhanced apoptosis, and down-regulated the expression of vascular endothelial growth factor (VEGF). The mechanisms thought to be responsible for these fucoidan-mediated effects are inhibition of the expression of Bcl-2 (Bcl-2 preserves the mitochondria integrity), survivin, extracellular signal-regulated kinases (ERKs), and VEGF, and an increase in caspase-3 activation.” AND Conclusion:
Many studies have explored the use of seaweed in the fight against several diseases, including colorectal and breast cancers. Various therapeutic compounds from seaweed are able to induce apoptosis through different pathways and molecular mechanisms. Several studies indicated that fucoidan was able to induce apoptosis, inhibit angiogenesis, and suppress lung metastasis of breast cancer in vitro and in vivo [21,22,23,24,31,70]. Furthermore, fucoidan inhibited growth and induced apoptosis of HT-29 colon cancer cells . Laminarin induced apoptosis through the Fas and IGF-IR signaling pathways and through the intrinsic apoptotic and ErbB pathways . This review highlights the importance of seaweed in the fight against colorectal and breast cancer. SOURCE:
DIM / IC3 / ZINC / Stinging nettle root tea / instead of Tamoxifen******
Iodine for cancer:*******************************************************
Natural Products as aromatase inhibitors:
PDF file for essential oils:
Flax seeds: See cancer superfoods link under flax seeds
Ruth Lupu Ph D Cancer Researcher: **
Fenbendazole & supplements a cancer cure? ***************************
For much more on this subject look here:
Repurposed drugs – Disulfiram (Antabuse)
Stress & negative thoughts:
Please listen from min. mark 12 to 16
Min mark 6:20 – 9:02
Castor oil packs:
How to Use Castor Oil Packs to Help Dissolve Growths and Heal Scar Tissue
Figs or potatoes for breast lumps:
Ways to Inhibit IGF-1
If you have high IGF-1 levels, you might want to focus a bit more on things that inhibit IGF-1, especially if you’re genetically predisposed to cancer.
- Fasting , especially prolonged fasting 
- Carbs – A Higher dietary proportion of carbohydrates is associated with lower IGF-1 
- Protein restriction 
- Calorie restriction 
- Intense walking 
- Legumes 
- Royal jelly 
- Glucosamine 
- Bilberry 
- Luteolin 
- Curcumin 
- Resveratrol (Grapes – especially the skin, mulberries, peanuts) . Inhibits IGF-1 in intestinal cells, reducing collagen formation that might otherwise lead to scarring and narrowing of the intestinal passageway. (Resveratrol increases IGF-1 in the brain – see below).
- Apigenin [108, 109] – in a study of human prostate cancer
- Lycopene (Tomatoes, guava, rosehip, watermelon, papaya, apricot, pink grapefruit) 
- EGCG (Green tea) 
- Boron (fruits & vegetables, raisins, nuts, legumes) – reduces free IGF 
- Genistein (Soybeans and soy products, red clover, and Sicilian pistachio) 
- Inflammation: Histamine , TNF-a , IL-6 , IL-1-alpha  – the negative effect of IL-6 on muscle function may be caused by lower IGF-1 
Dec 28, 2018 – Flavonoids have gained importance as anti-cancer agents and have shown great ….. Galangin in combination with berberine synergistically led to cell cycle …. Chrysin was reported to be the most potent flavonoid functioning in the …… Liu R.H. Nutrition, and cancer potential synergy of phytochemicals in …
Conclusion: Berberine alone and in combination with doxorubicin inhibited cell proliferation, induced apoptosis and altered cell cycle distribution of breast cancer cells. Therefore, berberine showed to be a good candidate for further studies as a new anticancer drug in the treatment of human breast cancer.
Cervical cancer is the second most frequent cancer in women worldwide, yet in Turkey it ranks only ninth, which is 2 to 5 times lower than in Europe and North America.101 Although the reasons for this difference surely involve a combination of factors, including sociocultural differences, lack of population-based screening programs, or a lower human papillomavirus (HPV) prevalence rate in Turkey, it has been suggested that the low incidence of cervical cancer in Turkey correlates with its boron-rich soil.102 HPVs are the main cause of cervical cancer. HPV-16 and HPV-18 cause approximately 95% of all cervical cancers, and boron interferes in the life cycle of HPV.
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